Perforin-dependent CD4+ T-cell cytotoxicity contributes to control a murine poxvirus infection.

نویسندگان

  • Min Fang
  • Nicholas A Siciliano
  • Adam R Hersperger
  • Felicia Roscoe
  • Angela Hu
  • Xueying Ma
  • Ahamed R Shamsedeen
  • Laurence C Eisenlohr
  • Luis J Sigal
چکیده

CD4(+) T cells are generally regarded as helpers and regulators of the immune response. Although cytolytic CD4(+) T cells have been described, whether those generated during the course of a viral infection play a role in virus control remains unknown. Here we show that during acute infection with ectromelia virus, the mouse homolog of the human virus of smallpox, large numbers of CD4(+) T cells in the draining lymph node and liver of resistant mice have a cytotoxic phenotype. We also show that these cells kill targets in vivo in a perforin-dependent manner and that mice with specific deficiency of perforin in CD4(+) T cells have impaired virus control. Thus, perforin-dependent CD4(+) T-cell killing of infected cells is an important mechanism of antiviral defense.

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عنوان ژورنال:
  • Proceedings of the National Academy of Sciences of the United States of America

دوره 109 25  شماره 

صفحات  -

تاریخ انتشار 2012